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To the cell surface (Fig. 7). Continued signaling through TLR4 is important for NF- B-mediated gene expression and secretion of pro-inflammatory cytokines in macrophages during a pathogenic challenge (9). Here we show that stathmin can modulate this sustained signaling, likely by affecting the stability of MT to enhance intracellular trafficking of receptor-containing vesicles, MMPs, and cytokines

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